NEC Mice & Touchpads Driver



NEC Mice & Touchpads Driver

Bus mouse
A Microsoft InPort bus mouse adapter, in the form of an 8-bit ISA (XT-bus) card.
TypeComputer mouse input port
Production history
DesignerMicrosoft
Designedlate 1980s
Produced1980s to 2000
Superseded byPS/2 port, USB (2000; 21 years ago)
General specifications
ExternalYes
Cable9 wires plus shield
Pins9
ConnectorMini-DIN-9
Data
Data signal30–200 Hz (interrupt mode) with 3 button state signals and quadrature signals for mouse movement
Pin out
Female port pin layout from the front
Pin 1SW2Mouse button 2
Pin 2SW3Mouse button 3
Pin 3GNDGround
Pin 4XBX position
Pin 5YAY position
Pin 6YBY position
Pin 7SW1Mouse button 1
Pin 8Vcc+5 V Power
Pin 9XAX position
XA/XB and YA/YB indicate movement and direction based on quadrature phase.

NEC was induced in C57BL/6 mice between P5 and P9 by hyperosmolar formula gavage feeding, oral LPS and exposure to systemic hypoxia, according to an established protocol (Zani et al., 2016). To minimize potential effects of undernutrition in our analysis, pups were fed 50 µl/g three times a day, at 07:00, 14:00 and 22:00. Nec 5133 MultiMedia Keyboard Nec Mini Mouse MS-Mini-01 Welcome to the HelpDrivers, driver for keyboards Nec. HelpDrivers offers drivers that support both currently shipping and obsolete keyboards, which are only available from this site. © New England College Athletics. 98 Bridge Street Henniker, N.H. 03242 603-428-2000 Privacy Policy.

A bus mouse is a variety of PCcomputer mouse which is attached to the computer using a specialized interface (originally, the Microsoft InPort interface developed for Microsoft's original mouse product).

Microsoft InPort™ bus mouse, showing the 9-pin round connector
Label on the bottom of a Microsoft InPort™ bus mouse, showing the FCC ID 'C3K7PN9937'

In the late 1980s, mice were not integrated with IBM-compatible personal computers, and the specialized bus interface (implemented via an ISA add-in card) was one of two popular ways to connect a mouse. Download primera usb devices driver windows 7. (Serial interfaces, common on engineering workstations, were the other method.) When the IBM PS/2 was introduced, it included a motherboard mouse interface which was integrated with the keyboard controller (still called the PS/2 mouse interface long after the PS/2 brand was withdrawn); this fairly quickly drove the bus mouse design out of the marketplace.

The bus mouse lived on in the NEC PC-98 family of personal computers in Japan.

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Touchpads

See also[edit]

Further reading[edit]

  • Paul, Matthias R. (2002-04-06). 'Re: [fd-dev] ANNOUNCE: CuteMouse 2.0 alpha 1'. freedos-dev. Archived from the original on 2020-02-07. Retrieved 2020-02-07. […] The original Mouse Systems Bus Mouse is a normal serial 8250 compatible mouse using the normal Mouse Systems serial protocol, however the base address of this 8250 type chip is not one of the usual COM port addresses 3F8h, 2F8h, 3E8h, or 2E8h, but either 238h or 338h. Besides others these addresses are also supported as alternative addresses for serial ports on the German c't UniRAM add-on ISA card. […] Bus mice from other vendors use completely different interfaces, partially residing at the same I/O addresses […]

External links[edit]

  • 'Mouse Connector'. Archived from the original on 2010-07-31. Retrieved 2006-10-27.
Retrieved from 'https://en.wikipedia.org/w/index.php?title=Bus_mouse&oldid=939595835'

NEC is likely a NETs dependent process and markers of NETosis are predictive of NEC in mice and humans

Nec Mice & Touchpads Driver Downloads

Mice

Nec Mice & Touchpads Driver Download

Abstract

Necrotizing enterocolitis (NEC) is one of the most devastating diseases affecting premature and mature infants. It is hypothesized that NEC is the result of neutrophils' active role in hyperinflammation after bacterial gut colonization, through their nuclear DNA release and formation of neutrophil extracellular traps (NETs) to combat pathogens. The aim of this study was to evaluate the importance of NETs in NEC pathogenesis, as well as to identify and validate markers of NETosis to predict NEC. NEC was induced in mice by gavage feeding of Neocate and lipopolysaccharide, followed by ten minutes of hypoxia (5% O2) q12h for five days, starting on day four postpartum (p.p.). The interrelation of NEC and neutrophils, including NETs, was assessed macroscopically (i.e. NEC score, SYTOX Orange), microscopically (i.e. Chiu score, citrullinated histone H3, neutrophil elastase), and in blood samples (i.e. cell-free DNA (cfDNA), DNase). In order to determine the exact role of NETs in NEC pathogenesis, a protein arginine deiminase (PAD) inhibition model was established (preventing NETs formation in mice) by injecting BB-Cl-amidine once daily, starting on day one p.p. Additionally, human intestinal samples of diagnostically verified NEC were analyzed. In total, 76 mice were analyzed in the experiment. Serum cfDNA correlated positively with NEC manifestation, as measured by macroscopic NEC score (r = 0.53, p = 0.001), and microscopic evaluation with Chiu score (r = 0.56, p < 0.001). Markers of neutrophil activation and NETosis were significantly increased in animals with NEC and in human samples as compared to controls. Further, prevention of NETosis by protein arginine deiminase (PAD) inhibition in mice significantly reduced mortality, tissue damage, and inflammation in mice induced with NEC. Our results suggest that the hyperinflammation observed in NEC is a NETs-dependent process, as NEC severity was significantly reduced in mice incapable of forming NETs (PAD inhibition) and markers for NEC and NETs correlated positively during the time course of NEC induction. Further, serum surrogate markers of NETosis (such as cfDNA and DNase) appear to predict NEC in neonatal mice. As findings of the mouse NEC model correlate positively with human NEC samples immunohistochemically, the hyperinflammation reaction observed in mice could potentially be applied to human NEC pathogenesis.

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Nec Mice & Touchpads Driver Update

Publication:
Pub Date:
August 2018
DOI:
10.1038/s41598-018-31087-0
Bibcode:
2018NatSR..812612V